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According to the activation synthesis theory dreaming PSYC

He also believed the instigating force behind dreams was always an instinctual and unconscious wish.

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Dreams are the language of a person's subconscious mind.

Photosynthesis, for example, is the concerted action of dozens of proteins (genes) with copy numbers in the hundreds to enable a simple chemical equation: carbon dioxide + water = sugar.

Freud described a number of different  that the mind uses to censor the latent content of a dream.

An increase in the permeability of outer mitochondrial membrane is central to apoptotic cell death, and results in the release of several apoptogenic factors such as cytochrome c into the cytoplasm to activate downstream destructive programs. The voltage-dependent anion channel (VDAC or mitochondrial porin) plays an essential role in disrupting the mitochondrial membrane barrier and is regulated directly by members of the Bcl-2 family proteins. Anti-apoptotic Bcl-2 family members interact with and close the VDAC, whereas some, but not all, proapoptotic members interact with VDAC to open protein-conducting pore through which apoptogenic factors pass. Although the VDAC is involved directly in breaking the mitochondrial membrane barrier and is a known component of the permeability transition pore complex, VDAC-dependent increase in outer membrane permeability can be independent of the permeability transition event such as mitochondrial swelling followed by rupture of the outer mitochondrial membrane. VDAC interacts not only with Bcl-2 family members but also with proteins such as gelsolin, an actin regulatory protein, and appears to be a convergence point for a variety of cell survival and cell death signals

Often, the waking mind recalls dreams only vaguely, if at all.

This find contradicts the classic Freudian theory of a driving force behind all dreams.

The regulatory sequences allow a cell to control when and how a gene is expressed its gene product (= RNA or protein) encoded by the coding sequence is synthesized.

Ramer’s group at the Unviersity of Rostock in Germany has done excellent work on CBD use in lung cancer with a 2012 published in FASEB. They found early-onset upregulation (four-fold) of ICAM-1 (intercellular adhesion molecule-1) via cannabinoid receptors in CDB treated lung cancer cells. Later, 48 hrs on, they found upregulation of the tissue inhibitor of matrix metalloproteinases-1 (TIMP-1) accounting for the loss of invasiveness of the lung cancer cells. In-vivo experiments injecting lung cancer cells (A549, H358, and H460) into mice then treated with CBD showed 2-3 fold increase in ICAM-1 and TIMP-1 protein which decreased cancer cell invasiveness. Upon microscopic inspection, the number of lung metastatic lesions had been reduced in half in the CBD treated mice. In yet another study, Ramers group also a new mechanism, the downregulation of the plasminogen activator inhibitor PAI-1, a protein involved in tumor invasiveness.

There are many cellular mechanisms involved with protein synthesis.

To summarize, the activation synthesis theory essentially made three key assumptions:

After their discovery, the two known cannabinoid receptors, CB(1) and CB(2), have been the focus of research into the cellular signalling mechanisms of cannabinoids. The initial assessment, mainly derived from expression studies, was that cannabinoids, via G(i/o) proteins, negatively modulate cyclic AMP levels, and activate inward rectifying K(+) channels. Recent findings have complicated this assessment on different levels: (1) cannabinoids include a wide range of compounds with varying profiles of affinity and efficacy at the known CB receptors, and these profiles do not necessarily match their biological activity; (2) CB receptors appear to be intrinsically active and possibly coupled to more than one type of G protein; (3) CB receptor signalling mechanisms are diverse and dependent on the system studied; (4) cannabinoids have other targets than CB receptors. The aim of this mini review is to discuss the current literature regarding CB receptor signalling pathways. These include regulation of adenylyl cyclase, MAP kinase, intracellular Ca(2+), and ion channels. In addition, actions of cannabinoids that are not mediated by CB(1) or CB(2) receptors are discussed.

In the present work, we investigated how the CB1receptor is coupled to ERK activation and its implications in cannabinoid regulation of glial cell death/survival decision.

In the start of the dream I was dreaming and woke up to write down that dream in the journal.
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    The main emphasis of the Activation-synthesis theory is dreaming is not psychological but physiological.

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    Unfortunately, I could not remember the dream and I felt stressed and pressured to have an interesting dream.

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Harvard psychiatrists J. Allan Hobson and Robert McCarley first proposed their theory in 1977, suggesting that dreaming results from the brain's attempt to make sense of neural activity that takes place during sleep.

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The activation-synthesis model suggests that dreams are caused by the physiological processes of the brain. While people used to believe that sleeping and dreaming was a passive process, researchers now know that the brain is anything but quiet during sleep. A wide variety of neural activity takes place as we slumber. Part of this is because sleep helps the brain perform a number of activities including and consolidating memories from the previous day.

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Hobson and McCarley see our poor ability to recall our dreams as reflecting "a state-dependent amnesia, since a carefully effected state change, to waking, may produce abundant recall even of highly charged dream material." So with that logic in mind if you are rapidly awakened out of REM sleep, you are likely to remember dreams that you would otherwise forget.

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This paper published in the American Journal of Psychiatry suggested that the occurrence of dreaming sleep is physiologically determined by a "dream state generator" located in the brain stem.

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According to Hobson and other researchers, circuits in the brain stem are activated during REM sleep. Once these circuits are activated, areas of the limbic system involved in emotions, sensations, and memories, including the amygdala and , become active. The brain synthesizes and interprets this internal activity and attempts create meaning from these signals, which results in dreaming.

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